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05:50, 7 December 2019 DixieChick (Talk | contribs) deleted page Y (eleven), are more inclined to fibrosis following harm (twelve). Another substantial gene (Spam: content was: "Provided that CSMD1 may be a fetal membranebound complement regulator, it may well provide a mechanism of fetomaternal tolerance for the duration of advance..." (and the only contributor was "[[Special:Contributions/Nyloniraq7|Nylon...)